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Gliotransmission and adenosine signaling promote axon regeneration

Wang, Fei
Ruppell, Kendra Takle
Zhou, Songlin
Qu, Yun
Gong, Jiaxin
Shang, Ye
Wu, Jinglin
Liu, Xin
Diao, Wenlin
Li, Yi
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Abstract

How glia control axon regeneration remains incompletely understood. Here, we investigate glial regulation of regenerative ability differences of closely related Drosophila larval sensory neuron subtypes. Axotomy elicits Ca2+ signals in ensheathing glia, which activates regenerative neurons through the gliotransmitter adenosine and mounts axon regenerative programs. However, non-regenerative neurons do not respond to glial stimulation or adenosine. Such neuronal subtype-specific responses result from specific expressions of adenosine receptors in regenerative neurons. Disrupting gliotransmission impedes axon regeneration of regenerative neurons, and ectopic adenosine receptor expression in non-regenerative neurons suffices to activate regenerative programs and induce axon regeneration. Furthermore, stimulating gliotransmission or activating the mammalian ortholog of Drosophila adenosine receptors in retinal ganglion cells (RGCs) promotes axon regrowth after optic nerve crush in adult mice. Altogether, our findings demonstrate that gliotransmission orchestrates neuronal subtype-specific axon regeneration in Drosophila and suggest that targeting gliotransmission or adenosine signaling is a strategy for mammalian central nervous system repair.

Source

Wang F, Ruppell KT, Zhou S, Qu Y, Gong J, Shang Y, Wu J, Liu X, Diao W, Li Y, Xiang Y. Gliotransmission and adenosine signaling promote axon regeneration. Dev Cell. 2023 Apr 24;58(8):660-676.e7. doi: 10.1016/j.devcel.2023.03.007. Epub 2023 Apr 6. PMID: 37028426.

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10.1016/j.devcel.2023.03.007
PubMed ID
37028426
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