TNFalpha-Mediated Cytotoxic Responses to IAP Inhibition Are Limited by the p38alpha MAPK Pathway
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UMass Chan Affiliations
Program in Molecular MedicineDocument Type
Journal ArticlePublication Date
2016-02-08
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Smac mimetics (SMs), a class of drugs that can promote tumor cell death, represent a potential therapeutic strategy for the treatment of cancer. In this issue of Cancer Cell, Lalaoui et al. (2016) report that SM efficacy can be potently increased by inhibition of the p38alpha MAPK/MK2 signaling pathway.Source
Cancer Cell. 2016 Feb 8;29(2):131-3. doi: 10.1016/j.ccell.2016.01.008. Link to article on publisher's siteDOI
10.1016/j.ccell.2016.01.008Permanent Link to this Item
http://hdl.handle.net/20.500.14038/28290PubMed ID
26859448Related Resources
Link to Article in PubMedae974a485f413a2113503eed53cd6c53
10.1016/j.ccell.2016.01.008