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dc.contributor.authorGirnius, Nomeda
dc.contributor.authorDavis, Roger J.
dc.date2022-08-11T08:08:16.000
dc.date.accessioned2022-08-23T15:48:48Z
dc.date.available2022-08-23T15:48:48Z
dc.date.issued2016-02-08
dc.date.submitted2016-02-19
dc.identifier.citationCancer Cell. 2016 Feb 8;29(2):131-3. doi: 10.1016/j.ccell.2016.01.008. <a href="http://dx.doi.org/10.1016/j.ccell.2016.01.008">Link to article on publisher's site</a>
dc.identifier.issn1535-6108 (Linking)
dc.identifier.doi10.1016/j.ccell.2016.01.008
dc.identifier.pmid26859448
dc.identifier.urihttp://hdl.handle.net/20.500.14038/28290
dc.description.abstractSmac mimetics (SMs), a class of drugs that can promote tumor cell death, represent a potential therapeutic strategy for the treatment of cancer. In this issue of Cancer Cell, Lalaoui et al. (2016) report that SM efficacy can be potently increased by inhibition of the p38alpha MAPK/MK2 signaling pathway.
dc.language.isoen_US
dc.relation<a href="http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=pubmed&cmd=Retrieve&list_uids=26859448&dopt=Abstract">Link to Article in PubMed</a>
dc.relation.urlhttp://dx.doi.org/10.1016/j.ccell.2016.01.008
dc.subjectBiochemistry
dc.subjectCell Biology
dc.subjectCellular and Molecular Physiology
dc.subjectMolecular Biology
dc.titleTNFalpha-Mediated Cytotoxic Responses to IAP Inhibition Are Limited by the p38alpha MAPK Pathway
dc.typeJournal Article
dc.source.journaltitleCancer cell
dc.source.volume29
dc.source.issue2
dc.identifier.legacycoverpagehttps://escholarship.umassmed.edu/davis/20
dc.identifier.contextkey8179702
html.description.abstract<p>Smac mimetics (SMs), a class of drugs that can promote tumor cell death, represent a potential therapeutic strategy for the treatment of cancer. In this issue of Cancer Cell, Lalaoui et al. (2016) report that SM efficacy can be potently increased by inhibition of the p38alpha MAPK/MK2 signaling pathway.</p>
dc.identifier.submissionpathdavis/20
dc.contributor.departmentUMass Metabolic Network
dc.contributor.departmentProgram in Molecular Medicine
dc.source.pages131-3


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