Show simple item record

dc.contributor.authorLeidner, Florian
dc.contributor.authorYilmaz, Nese Kurt
dc.contributor.authorSchiffer, Celia A.
dc.date2022-08-11T08:08:28.000
dc.date.accessioned2022-08-23T15:56:16Z
dc.date.available2022-08-23T15:56:16Z
dc.date.issued2021-06-28
dc.date.submitted2022-01-02
dc.identifier.citation<p>Leidner F, Kurt Yilmaz N, Schiffer CA. Deciphering Antifungal Drug Resistance in <em>Pneumocystis jirovecii</em> DHFR with Molecular Dynamics and Machine Learning. J Chem Inf Model. 2021 Jun 28;61(6):2537-2541. doi: 10.1021/acs.jcim.1c00403. Epub 2021 Jun 17. PMID: 34138546. <a href="https://doi.org/10.1021/acs.jcim.1c00403">Link to article on publisher's site</a></p>
dc.identifier.issn1549-9596 (Linking)
dc.identifier.doi10.1021/acs.jcim.1c00403
dc.identifier.pmid34138546
dc.identifier.urihttp://hdl.handle.net/20.500.14038/29931
dc.description.abstractDrug resistance impacts the effectiveness of many new therapeutics. Mutations in the therapeutic target confer resistance; however, deciphering which mutations, often remote from the enzyme active site, drive resistance is challenging. In a series of Pneumocystis jirovecii dihydrofolate reductase variants, we elucidate which interactions are key bellwethers to confer resistance to trimethoprim using homology modeling, molecular dynamics, and machine learning. Six molecular features involving mainly residues that did not vary were the best indicators of resistance.
dc.language.isoen_US
dc.relation<p><a href="http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=pubmed&cmd=Retrieve&list_uids=34138546&dopt=Abstract">Link to Article in PubMed</a></p>
dc.relation.urlhttps://doi.org/10.1021/acs.jcim.1c00403
dc.subjectDrug resistance
dc.subjectPeptides and proteins
dc.subjectGenetics
dc.subjectAntifungal activit
dc.subjectBiochemistry, Biophysics, and Structural Biology
dc.subjectMedicinal and Pharmaceutical Chemistry
dc.subjectMedicinal Chemistry and Pharmaceutics
dc.subjectMedicinal-Pharmaceutical Chemistry
dc.titleDeciphering Antifungal Drug Resistance in Pneumocystis jirovecii DHFR with Molecular Dynamics and Machine Learning
dc.typeJournal Article
dc.source.journaltitleJournal of chemical information and modeling
dc.source.volume61
dc.source.issue6
dc.identifier.legacycoverpagehttps://escholarship.umassmed.edu/faculty_pubs/2134
dc.identifier.contextkey27074125
html.description.abstract<p>Drug resistance impacts the effectiveness of many new therapeutics. Mutations in the therapeutic target confer resistance; however, deciphering which mutations, often remote from the enzyme active site, drive resistance is challenging. In a series of Pneumocystis jirovecii dihydrofolate reductase variants, we elucidate which interactions are key bellwethers to confer resistance to trimethoprim using homology modeling, molecular dynamics, and machine learning. Six molecular features involving mainly residues that did not vary were the best indicators of resistance.</p>
dc.identifier.submissionpathfaculty_pubs/2134
dc.contributor.departmentSchiffer Lab
dc.contributor.departmentDepartment of Biochemistry and Molecular Pharmacology
dc.source.pages2537-2541


This item appears in the following Collection(s)

Show simple item record