Show simple item record

dc.contributor.authorCorvera, Silvia
dc.contributor.authorBurkart, Alison
dc.contributor.authorKim, Ja-Young
dc.contributor.authorChristianson, Jennifer L.
dc.contributor.authorWang, Zhao
dc.contributor.authorScherer, Philipp E.
dc.date2022-08-11T08:08:56.000
dc.date.accessioned2022-08-23T16:13:15Z
dc.date.available2022-08-23T16:13:15Z
dc.date.issued2006-08-17
dc.date.submitted2008-08-27
dc.identifier.citationGenes Dev. 2006 Aug 15;20(16):2193-201. <a href="http://dx.doi.org/10.1101/gad.1447506">Link to article on publisher's site</a>
dc.identifier.issn0890-9369 (Print)
dc.identifier.doi10.1101/gad.1447506
dc.identifier.pmid16912272
dc.identifier.urihttp://hdl.handle.net/20.500.14038/33579
dc.description.abstractThe dysregulation of specific cellular functions in adipocytes, muscle cells, beta cells, and the liver leads to changes in systemic metabolic processes and ultimately to the pathophysiological manifestations that cause type 2 diabetes. The underlying cellular mechanisms are complex. The two meetings summarized here aimed to highlight the recent advances in our understanding of the molecular basis of feeding and nutrient storage and on the molecular consequences of obesity in terms of promoting risk for type 2 diabetes and cardiovascular disease.
dc.language.isoen_US
dc.relation<a href="http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=16912272&dopt=Abstract ">Link to article in PubMed</a>
dc.relation.urlhttp://dx.doi.org/10.1101/gad.1447506
dc.subjectAdipogenesis; Animals; Diabetes Mellitus; Energy Metabolism; Humans; Inflammation; Obesity; Signal Transduction
dc.subjectBiochemistry, Biophysics, and Structural Biology
dc.subjectLife Sciences
dc.subjectMedicine and Health Sciences
dc.titleKeystone meeting summary: 'Adipogenesis, obesity, and inflammation' and 'Diabetes mellitus and the control of cellular energy metabolism, ' January 21-26, 2006, Vancouver, Canada
dc.typeJournal Article
dc.source.journaltitleGenes and development
dc.source.volume20
dc.source.issue16
dc.identifier.legacycoverpagehttps://escholarship.umassmed.edu/gsbs_sp/249
dc.identifier.contextkey606047
html.description.abstract<p>The dysregulation of specific cellular functions in adipocytes, muscle cells, beta cells, and the liver leads to changes in systemic metabolic processes and ultimately to the pathophysiological manifestations that cause type 2 diabetes. The underlying cellular mechanisms are complex. The two meetings summarized here aimed to highlight the recent advances in our understanding of the molecular basis of feeding and nutrient storage and on the molecular consequences of obesity in terms of promoting risk for type 2 diabetes and cardiovascular disease.</p>
dc.identifier.submissionpathgsbs_sp/249
dc.contributor.departmentProgram in Molecular Medicine
dc.source.pages2193-201
dc.contributor.studentAlison Burkart; Jennifer L. Christianson


This item appears in the following Collection(s)

Show simple item record