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dc.contributor.authorSilverman, Neal S.
dc.contributor.authorManiatis, Tom
dc.date2022-08-11T08:09:08.000
dc.date.accessioned2022-08-23T16:18:43Z
dc.date.available2022-08-23T16:18:43Z
dc.date.issued2001-09-15
dc.date.submitted2008-12-19
dc.identifier.citationGenes Dev. 2001 Sep 15;15(18):2321-42. <a href="http://dx.doi.org/10.1101/gad.909001">Link to article on publisher's site</a>
dc.identifier.issn0890-9369 (Print)
dc.identifier.doi10.1101/gad.909001
dc.identifier.pmid11562344
dc.identifier.urihttp://hdl.handle.net/20.500.14038/34881
dc.description<p>At the time of publication, Neal Silverman was not yet affiliated with the University of Massachusetts Medical School.</p>
dc.description.abstractIn this review, we discuss recent advances in understanding the signaling pathways in mammalian and Drosophila innate immunity, with emphasis on the mechanisms by which NF-kappaB/Rel family proteins are activated.
dc.language.isoen_US
dc.relation<a href="http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=pubmed&cmd=Retrieve&list_uids=11562344&dopt=Abstract">Link to Article in PubMed</a>
dc.relation.urlhttp://dx.doi.org/10.1101/gad.909001
dc.rightsPublisher PDF posted as allowed by the publisher's author rights policy at http://genesdev.cshlp.org/site/misc/terms.xhtml. Beginning six months from the full-issue publication date, articles published in Genes & Development that are not designated as Open Access are distributed under the Creative Commons Attribution-Non-Commercial 4.0 International License (CC-BY-NC), as described at http://creativecommons.org/licenses/by-nc/4.0/. This license permits non-commercial use, including reproduction, adaptation, and distribution of the article provided the original author and source are credited.
dc.rights.urihttp://creativecommons.org/licenses/by-nc/4.0/
dc.subjectInsects
dc.subjectNF-kappa B
dc.subjectPhylogeny
dc.subjectSignal Transduction
dc.subjectinnate immunity
dc.subjectsignaling pathways
dc.subjectDrosophila
dc.subjectNF-kappaB
dc.subjectImmunity
dc.subjectMolecular Biology
dc.titleNF-kappaB signaling pathways in mammalian and insect innate immunity
dc.typeJournal Article
dc.source.journaltitleGenes and development
dc.source.volume15
dc.source.issue18
dc.identifier.legacyfulltexthttps://escholarship.umassmed.edu/cgi/viewcontent.cgi?article=1010&amp;context=infdis_pp&amp;unstamped=1
dc.identifier.legacycoverpagehttps://escholarship.umassmed.edu/infdis_pp/11
dc.identifier.contextkey684322
refterms.dateFOA2022-08-23T16:18:43Z
html.description.abstract<p>In this review, we discuss recent advances in understanding the signaling pathways in mammalian and Drosophila innate immunity, with emphasis on the mechanisms by which NF-kappaB/Rel family proteins are activated.</p>
dc.identifier.submissionpathinfdis_pp/11
dc.contributor.departmentDepartment of Medicine, Division of Infectious Diseases and Immunology
dc.source.pages2321-42


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Publisher PDF posted as allowed by the publisher's author rights policy at http://genesdev.cshlp.org/site/misc/terms.xhtml. Beginning six months from the full-issue publication date, articles published in Genes & Development that are not designated as Open Access are distributed under the Creative Commons Attribution-Non-Commercial 4.0 International License (CC-BY-NC), as described at http://creativecommons.org/licenses/by-nc/4.0/. This license permits non-commercial use, including reproduction, adaptation, and distribution of the article provided the original author and source are credited.
Except where otherwise noted, this item's license is described as Publisher PDF posted as allowed by the publisher's author rights policy at http://genesdev.cshlp.org/site/misc/terms.xhtml. Beginning six months from the full-issue publication date, articles published in Genes & Development that are not designated as Open Access are distributed under the Creative Commons Attribution-Non-Commercial 4.0 International License (CC-BY-NC), as described at http://creativecommons.org/licenses/by-nc/4.0/. This license permits non-commercial use, including reproduction, adaptation, and distribution of the article provided the original author and source are credited.