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dc.contributor.authorLamech, Lilian
dc.contributor.authorHaynes, Cole M
dc.date2022-08-11T08:09:19.000
dc.date.accessioned2022-08-23T16:26:42Z
dc.date.available2022-08-23T16:26:42Z
dc.date.issued2017-11-16
dc.date.submitted2017-12-06
dc.identifier.citationMol Cell. 2017 Nov 16;68(4):641-642. doi: 10.1016/j.molcel.2017.11.003. <a href="https://doi.org/10.1016/j.molcel.2017.11.003">Link to article on publisher's site</a>
dc.identifier.issn1097-2765 (Linking)
dc.identifier.doi10.1016/j.molcel.2017.11.003
dc.identifier.pmid29149591
dc.identifier.urihttp://hdl.handle.net/20.500.14038/36622
dc.description.abstractMaintenance of mitochondrial function during stress conditions is vital for cellular survival. In this issue of Molecular Cell, Nielson et al. (2017) characterize a unique domain within Vms1 that allows this protein quality control component to specifically recognize damaged or stressed compartments within the mitochondrial network.
dc.language.isoen_US
dc.relation<a href="http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=pubmed&cmd=Retrieve&list_uids=29149591&dopt=Abstract">Link to Article in PubMed</a>
dc.relation.urlhttps://doi.org/10.1016/j.molcel.2017.11.003
dc.subjectBiochemistry
dc.subjectCell Biology
dc.subjectCellular and Molecular Physiology
dc.subjectMolecular Biology
dc.titleMitochondrial Sterol Oxidation Marks the Spot
dc.typeJournal Article
dc.source.journaltitleMolecular cell
dc.source.volume68
dc.source.issue4
dc.identifier.legacycoverpagehttps://escholarship.umassmed.edu/metnet_pubs/140
dc.identifier.contextkey11207793
html.description.abstract<p>Maintenance of mitochondrial function during stress conditions is vital for cellular survival. In this issue of Molecular Cell, Nielson et al. (2017) characterize a unique domain within Vms1 that allows this protein quality control component to specifically recognize damaged or stressed compartments within the mitochondrial network.</p>
dc.identifier.submissionpathmetnet_pubs/140
dc.contributor.departmentUMass Metabolic Network
dc.contributor.departmentDepartment of Molecular, Cell and Cancer Biology
dc.source.pages641-642


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