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dc.contributor.authorOlive, Andrew J.
dc.contributor.authorSassetti, Christopher M.
dc.date2022-08-11T08:09:20.000
dc.date.accessioned2022-08-23T16:27:00Z
dc.date.available2022-08-23T16:27:00Z
dc.date.issued2016-04-01
dc.date.submitted2017-04-20
dc.identifier.citationNat Rev Microbiol. 2016 Apr;14(4):221-34. Epub 2016 Mar 7. <a href="https://doi.org/10.1038/nrmicro.2016.12">Link to article on publisher's site</a>
dc.identifier.issn1740-1526 (Linking)
dc.identifier.doi10.1038/nrmicro.2016.12
dc.identifier.pmid26949049
dc.identifier.urihttp://hdl.handle.net/20.500.14038/36682
dc.description.abstractOur understanding of bacterial pathogenesis is dominated by the cell biology of the host-pathogen interaction. However, the majority of metabolites that are used in prokaryotic and eukaryotic physiology and signalling are chemically similar or identical. Therefore, the metabolic crosstalk between pathogens and host cells may be as important as the interactions between bacterial effector proteins and their host targets. In this Review we focus on host-pathogen interactions at the metabolic level: chemical signalling events that enable pathogens to sense anatomical location and the local physiology of the host; microbial metabolic pathways that are dedicated to circumvent host immune mechanisms; and a few metabolites as central points of competition between the host and bacterial pathogens.
dc.language.isoen_US
dc.relation<a href="http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=pubmed&cmd=Retrieve&list_uids=26949049&dopt=Abstract">Link to Article in PubMed</a>
dc.relation.urlhttps://doi.org/10.1038/nrmicro.2016.12
dc.subjectCellular and Molecular Physiology
dc.subjectMicrobiology
dc.titleMetabolic crosstalk between host and pathogen: sensing, adapting and competing
dc.typeJournal Article
dc.source.journaltitleNature reviews. Microbiology
dc.source.volume14
dc.source.issue4
dc.identifier.legacycoverpagehttps://escholarship.umassmed.edu/metnet_pubs/50
dc.identifier.contextkey10048071
html.description.abstract<p>Our understanding of bacterial pathogenesis is dominated by the cell biology of the host-pathogen interaction. However, the majority of metabolites that are used in prokaryotic and eukaryotic physiology and signalling are chemically similar or identical. Therefore, the metabolic crosstalk between pathogens and host cells may be as important as the interactions between bacterial effector proteins and their host targets. In this Review we focus on host-pathogen interactions at the metabolic level: chemical signalling events that enable pathogens to sense anatomical location and the local physiology of the host; microbial metabolic pathways that are dedicated to circumvent host immune mechanisms; and a few metabolites as central points of competition between the host and bacterial pathogens.</p>
dc.identifier.submissionpathmetnet_pubs/50
dc.contributor.departmentUMass Metabolic Network
dc.contributor.departmentDepartment of Microbiology and Physiological Systems
dc.source.pages221-34


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