An autophagy-independent function for Beclin 1 in cancer
| dc.contributor.author | Rohatgi, Rasika | |
| dc.contributor.author | Shaw, Leslie M. | |
| dc.date | 2022-08-11T08:09:20.000 | |
| dc.date.accessioned | 2022-08-23T16:27:02Z | |
| dc.date.available | 2022-08-23T16:27:02Z | |
| dc.date.issued | 2016-01-02 | |
| dc.date.submitted | 2017-04-20 | |
| dc.identifier.citation | Mol Cell Oncol. 2016 Jan 2;3(1). pii: e1030539. Epub 2015 Apr 14. <a href="https://doi.org/10.1080/23723556.2015.1030539">Link to article on publisher's site</a> | |
| dc.identifier.issn | 2372-3556 (Linking) | |
| dc.identifier.doi | 10.1080/23723556.2015.1030539 | |
| dc.identifier.pmid | 26998512 | |
| dc.identifier.uri | http://hdl.handle.net/20.500.14038/36691 | |
| dc.description.abstract | The involvement of Beclin 1 in cancer has been attributed primarily to its role in autophagy initiation. Our recent findings identify a novel, alternative mechanism by which Beclin 1 can impact tumor progression, through the regulation of growth factor receptor signaling. | |
| dc.language.iso | en_US | |
| dc.relation | <a href="http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=pubmed&cmd=Retrieve&list_uids=26998512&dopt=Abstract">Link to Article in PubMed</a> | |
| dc.relation.url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4792009/ | |
| dc.subject | autophagy | |
| dc.subject | beclin 1 | |
| dc.subject | cancer | |
| dc.subject | endocytosis | |
| dc.subject | growth factor receptor | |
| dc.subject | signaling | |
| dc.subject | Biochemistry | |
| dc.subject | Cancer Biology | |
| dc.subject | Cell Biology | |
| dc.subject | Cellular and Molecular Physiology | |
| dc.subject | Molecular Biology | |
| dc.title | An autophagy-independent function for Beclin 1 in cancer | |
| dc.type | Journal Article | |
| dc.source.journaltitle | Molecular and cellular oncology | |
| dc.source.volume | 3 | |
| dc.source.issue | 1 | |
| dc.identifier.legacycoverpage | https://escholarship.umassmed.edu/metnet_pubs/59 | |
| dc.identifier.contextkey | 10048081 | |
| html.description.abstract | <p>The involvement of Beclin 1 in cancer has been attributed primarily to its role in autophagy initiation. Our recent findings identify a novel, alternative mechanism by which Beclin 1 can impact tumor progression, through the regulation of growth factor receptor signaling.</p> | |
| dc.identifier.submissionpath | metnet_pubs/59 | |
| dc.contributor.department | UMass Metabolic Network | |
| dc.contributor.department | Department of Molecular, Cell and Cancer Biology |