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dc.contributor.authorKoupenova-Zamor, Milka
dc.contributor.authorKehrel, Beate E.
dc.contributor.authorCorkrey, Heather A.
dc.contributor.authorFreedman, Jane E.
dc.date2022-08-11T08:09:20.000
dc.date.accessioned2022-08-23T16:27:02Z
dc.date.available2022-08-23T16:27:02Z
dc.date.issued2017-03-14
dc.date.submitted2017-02-16
dc.identifier.citationEur Heart J. 2017 Mar 14;38(11):785-791. doi: 10.1093/eurheartj/ehw550. <a href="https://doi.org/10.1093/eurheartj/ehw550">Link to article on publisher's site</a>
dc.identifier.issn0195-668X (Linking)
dc.identifier.doi10.1093/eurheartj/ehw550
dc.identifier.pmid28039338
dc.identifier.urihttp://hdl.handle.net/20.500.14038/36692
dc.description.abstractHaemostasis and thrombosis are complex, multifactorial processes. There is an evolving understanding of the mechanisms influencing vascular occlusion and the role of inflammation and immunity. Despite major advances in elucidating the mechanistic pathways mediating platelet function and thrombosis, challenges in the treatment of vascular occlusive diseases persist. Pharmacological advances have greatly affected thrombotic outcomes, but this has led to the unwanted side effect of bleeding. Detailed assessment of the impact of non-thrombotic diseases on haemostasis and thrombosis is necessary to better evaluate thrombotic risk and establish optimal treatment. This review will focus on recent advances in understanding the contribution of evolving risk factors to thrombosis.
dc.language.isoen_US
dc.relation<a href="http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?db=pubmed&cmd=Retrieve&list_uids=28039338&dopt=Abstract">Link to Article in PubMed</a>
dc.relation.urlhttps://doi.org/10.1093/eurheartj/ehw550
dc.subjectPlatelets
dc.subjectThrombosis
dc.subjectCoagulation
dc.subjectCardiovascular
dc.subjectBleeding
dc.subjectBiochemistry
dc.subjectCardiology
dc.subjectCell Biology
dc.subjectCellular and Molecular Physiology
dc.subjectMolecular Biology
dc.titleThrombosis and platelets: an update
dc.typeJournal Article
dc.source.journaltitleEuropean heart journal
dc.source.volume38
dc.source.issue11
dc.identifier.legacycoverpagehttps://escholarship.umassmed.edu/metnet_pubs/6
dc.identifier.contextkey9698860
html.description.abstract<p>Haemostasis and thrombosis are complex, multifactorial processes. There is an evolving understanding of the mechanisms influencing vascular occlusion and the role of inflammation and immunity. Despite major advances in elucidating the mechanistic pathways mediating platelet function and thrombosis, challenges in the treatment of vascular occlusive diseases persist. Pharmacological advances have greatly affected thrombotic outcomes, but this has led to the unwanted side effect of bleeding. Detailed assessment of the impact of non-thrombotic diseases on haemostasis and thrombosis is necessary to better evaluate thrombotic risk and establish optimal treatment. This review will focus on recent advances in understanding the contribution of evolving risk factors to thrombosis.</p>
dc.identifier.submissionpathmetnet_pubs/6
dc.contributor.departmentUMass Metabolic Network
dc.contributor.departmentDepartment of Medicine, Division of Cardiovascular Medicine
dc.source.pages785-791


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