AuthorsGravallese, Ellen M.
UMass Chan AffiliationsDepartment of Medicine, Division of Rheumatology
Document TypeResponse or Comment
Disease Models, Animal
Mice, Inbred C57BL
Skin and Connective Tissue Diseases
MetadataShow full item record
AbstractThe molecular mechanisms underlying the putative role of osteopontin in the chronic inflammatory disease rheumatoid arthritis are unclear. A study in a murine model of arthritis now demonstrates that a specific antibody directed against the exposed osteopontin epitope SLAYGLR is capable of preventing inflammatory cell infiltration in arthritic joints.
SourceJ Clin Invest. 2003 Jul;112(2):147-9. Link to article on publisher's site
Permanent Link to this Itemhttp://hdl.handle.net/20.500.14038/48755
At the time of publication, Ellen Gravallese was not yet affiliated with the University of Massachusetts Medical School.
Related ResourcesLink to Article in PubMed
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