Gravallese, Ellen M.2022-08-232022-08-232003-07-172015-02-25J Clin Invest. 2003 Jul;112(2):147-9. <a href="http://dx.doi.org/10.1172/JCI19190">Link to article on publisher's site</a>0021-9738 (Linking)10.1172/JCI1919012865402https://hdl.handle.net/20.500.14038/48755<p>At the time of publication, Ellen Gravallese was not yet affiliated with the University of Massachusetts Medical School.</p>The molecular mechanisms underlying the putative role of osteopontin in the chronic inflammatory disease rheumatoid arthritis are unclear. A study in a murine model of arthritis now demonstrates that a specific antibody directed against the exposed osteopontin epitope SLAYGLR is capable of preventing inflammatory cell infiltration in arthritic joints.en-US<p>Publisher PDF posted as allowed by the publisher's author rights policy at http://static.the-jci.org/content_assets/admin/forms/jcicopyright.pdf.</p>AnimalsArthritis, RheumatoidDisease Models, AnimalEpitopesInflammationMiceMice, Inbred C57BLOsteopontinRNA, MessengerSialoglycoproteinsosteopontinrheumatoid arthritisCellsMedical ImmunologyMusculoskeletal DiseasesRheumatologySkin and Connective Tissue DiseasesResponse or Commenthttps://escholarship.umassmed.edu/cgi/viewcontent.cgi?article=1061&amp;context=rheumatology_pubs&amp;unstamped=1https://escholarship.umassmed.edu/rheumatology_pubs/626724718rheumatology_pubs/62